Cancer prevention in women

Micronutrient therapy

Compensate for hormonal imbalances
Phytoestrogens from soy and red clover have strong antioxidant and anticarcinogenic effects. They inhibit the development of cancer cells and can slow down the formation of blood vessels in tumors. In pre-menopausal woman with elevated estrogen level, phytohormones can reduce the risk of breast cancer and other hormone-related tumors due to their attenuating effect. Post-menopause, when the body's own estrogen is greatly reduced, phytohormones act as a weak estrogen substitute and help to counteract associated physical changes. During perimenopause, which is characterized by strong fluctuations in estrogen levels, phytohormones have an inhibitory or supportive effect, i.e. they are generally balancing. This alleviates the typical symptoms of menopause.
Increased antioxidative capacity
Beta carotenes are effective antioxidants that bind free peroxyl radicals and singlet oxygen thereby preventing lipid peroxidation by UVB, X-rays and elevated radiation. The different carotenoid forms have different points of effect, but can complement and strengthen each other in their functions.

Selenium can play a protective role in the early stages of carcinogenesis due to its antioxidant effect. The oxidative activation of procarcinogenic compounds can be minimized and DNA changes can be prevented.

Nutrigenetics

Micronutrient Recommended daily intake 
Folic Acid 800 µg
Vitamin E 100 mg
Vitamin A 1000 µg
Vitamin D 50 µg
black cumin oil 1-3 g
15-30 mg
10 billion reproducibility germs / day

Diagnostic tests

Available laboratory tests (Laboratory GANZIMMUN) Detailed information
Blood serum Vitamin D3 (Calcidiol) <Vitamin D in tumor prophylaxis
HPV-PCR (Human Papilloma Virus) HPV DNA detection, HPV "High risk types", HPV "Low risk types" Human papilloma virus
Estrogen metabolites in urine 2-, 4-, 16-hydroxy-estrone, 2/16-hydroxy-estrone ratio, 2- and 4-methoxy-estrone, methylation activity <Estronex
Female tumor markers (profile) CA125, CA15-3, CA19-9, CEA, p53-Auto-AB  
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